Nephrology in 30 DaysMcGraw Hill Professional, 2005 M03 4 - 500 páginas A concise review of the core principles and clinical entities associated with nephrology at the point of care Covers all major acute and chronic kidney diseases, from acid/base disturbances to stones to end stage renal disease. Features a concise, manageable format with recommended timeframes for mastering the content, case studies, and summary tables. Perfect for trainees and practicing non-specialists. |
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Página 150
... symptoms. Patients with primary hyperparathyroidism present with mild asymptomatic hypercalcemia incidentally discovered on routine laboratory examination. Severe hypercalcemia is associated with prominent neurologic and GI symptoms ...
... symptoms. Patients with primary hyperparathyroidism present with mild asymptomatic hypercalcemia incidentally discovered on routine laboratory examination. Severe hypercalcemia is associated with prominent neurologic and GI symptoms ...
Página 156
... Symptoms. The degree of hypocalcemia and rate of decline of the serum calcium concentration determine whether hypocalcemic symptoms occur. The point at which symptoms occur depends on multiple factors including pH, and whether other ...
... Symptoms. The degree of hypocalcemia and rate of decline of the serum calcium concentration determine whether hypocalcemic symptoms occur. The point at which symptoms occur depends on multiple factors including pH, and whether other ...
Página 323
Robert Reilly, Mark A. Perazella. Signs. and. Symptoms. Symptoms. Signs and symptoms experienced by the patient with urinary tract obstruction depend on the rapidity and degree of obstruction. If obstruction occurs suddenly as in ...
Robert Reilly, Mark A. Perazella. Signs. and. Symptoms. Symptoms. Signs and symptoms experienced by the patient with urinary tract obstruction depend on the rapidity and degree of obstruction. If obstruction occurs suddenly as in ...
Contenido
1 INTRODUCTION | 1 |
2 DISORDERS OF SODIUM BALANCE | 13 |
3 DISORDERS OF WATER BALANCE HYPO AND HYPERNATREMIA | 30 |
Derechos de autor | |
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abnormalities acid activity acute addition agents aldosterone anion arterial associated blood body bone calcium cause cells changes chronic clinical common commonly complete concentration creatinine decreased deposition develop diabetes diagnosis disorders distal diuretic drugs edema effects elevated evaluation examination excretion factors Figure fluid formation function glomerular glucose HCO3 hypertension hypokalemia impaired important increased infection initial injury involved KEY POINTS kidney disease leads less levels loop loss magnesium major measure mechanisms membrane meq/L metabolic acidosis metabolic alkalosis mg/dL nephron normal obstruction occurs organic patients phosphate phosphorus plasma potassium present pressure primary production progression protein proteinuria proximal pyuria reabsorption receptor reduce renal failure resistance response result risk secondary secretion seen serum setting severe sodium stones studies symptoms syndrome Table therapy tion tract treat treatment tubular tubule urinary urine volume